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- IRMS - Nicolae Testemitanu SUMPh
- 1. COLECȚIA INSTITUȚIONALĂ
- MedEspera: International Medical Congress for Students and Young Doctors
- MedEspera 2024
Please use this identifier to cite or link to this item:
http://hdl.handle.net/20.500.12710/28686
Title: | Benign tumors of the vocal cords. General notions. Epidemiology, etiopathogenesis and treatment |
Authors: | Țîbulac Daniela |
Issue Date: | 2024 |
Publisher: | Instituţia Publică Universitatea de Stat de Medicină şi Farmacie „Nicolae Testemiţanu” din Republica Moldova |
Citation: | Țîbulac Daniela. Benign tumors of the vocal cords. General notions. Epidemiology, etiopathogenesis and treatment. In: Abstract Book. MedEspera 2024. The 10th International Medical Congress for Students and Young Doctors. 24-27 April 2024, Chișinău, Republic of Moldova, p. 275. ISBN 978-9975-3544-2-4. |
Abstract: | Introduction. Benign vocal lesions are non-malignant growths of abnormal tissue on the vocal cords. The common benign lesions of vocal cord are singer’s nodule, polyps, papilloma, polypoid degeneration (Reinke’s edema) and cysts. Others are sulcus vocalis, mucosal bridge, intracordal cysts, vocal cord varices and anterior webs. Several factors can be responsible for the development of the benign vocal lesions such as vocal abuse, overuse or misuse of voice, chronic infections of upper airway, allergy, smoking and gastroesophageal reflux. Frequent coughing and throat clearing also contribute to the mucosal irritation which worsens the voice. Excessive mechanical trauma and stress in the mid membranous area of the vocal cord leads to wound formation. Subsequently remodeling of the superficial layer of the lamina propria and, to a lesser extent, epithelium results in the formation of vocal cord nodules, polyps, and cysts. Several studies have demonstrated that the pathologic changes in vocal cord polyps, nodules, and cysts occur within the superficial layer of the lamina propria Aim of study. The etiological relationship between smoking and vocal fold polyps has been widely studied. Some authors consider smoking as the primary factor for the development of vocal fold polyps, mainly when associated with vocal abuse. Methods and materials. The present study is a review article based on a bibliographic search using platforms, databases and search engines, without restrictions on means of publication, methodological quality or language. Articles on benign vocal cord tumors related to the study object published in the last 5 years were included. Results. Vocal trauma, especially in women and children, is a strong risk factor for vocal cord nodules and cysts. Gastroesophageal reflux is a suggested risk factor for Reinke's edema and contact granuloma. Smoking and alcohol consumption are the risk factors that predominate in the male gender causing especially polyps and Reinke's edema. Identifying risk factors is an important step in therapy planning. Conclusion. The voice is the main actor in our daily life. It allows us the main means of communication between people. Approximately 30% of the population has dysphonia at some point in their lives. Since dysphonia is only a symptom and not a diagnosis, each case of dysphonia must be carefully examined to determine the causes. Also, early diagnosis of benign tumor processes is very important for treatment and restoration of the voice as quickly as possible. cords. The common benign lesions of vocal cord are sin ger’s nodule, polyps, papilloma, polypoid degeneration (Reinke’s edema) and cysts. Others are sulcus vocalis, mucosal bridge, intracordal cysts, vocal cord varices and anterior webs. Several fa ctors can be responsible for the development of the benign vocal lesions such as vocal abuse, over use or misuse of voice, chronic infections of upper airway, allergy, smoking and gastroesophageal reflux. Frequent coughing and throat clearing also contribute to the mucosal irritation which wor sens the voice. Excessive mechanical trauma and stress in the mid membranous area of the voca l cord leads to wound formation. Subsequently remodeling of the superficial layer of the lami na propria and, to a lesser extent, epithelium results in the formation of vocal cord nodul es, polyps, and cysts. Several studies have demonstrated that the pathologic changes in vocal cord pol yps, nodules, and cysts occur within the superficial layer of the lamina propria Aim of study. The etiological relationship between smoking and vocal f old polyps has been widely studied. Some authors consider smoking as the primary fac tor for the development of vocal fold polyps, mainly when associated with vocal abuse. Methods and materials. The present study is a review article based on a bibliographi c search using platforms, databases and search engines, without res trictions on means of publication, methodological quality or language. Articles on benign voc al cord tumors related to the study object published in the last 5 years were included. Results. Vocal trauma, especially in women and children, is a strong r isk factor for vocal cord nodules and cysts. Gastroesophageal reflux is a suggested ri sk factor for Reinke's edema and contact granuloma. Smoking and alcohol consumption are the risk factors that predominate in the male gender causing especially polyps and Reinke's edema. Identifyi ng risk factors is an important step in therapy planning. Conclusion. The voice is the main actor in our daily life. It allo ws us the main means of communication between people. Approximately 30% of the populatio n has dysphonia at some point in their lives. Since dysphonia is only a symptom a nd not a diagnosis, each case of dysphonia must be carefully examined to determine the causes. Also, ea rly diagnosis of benign tumor processes is very important for treatment and restoration o f the voice as quickly as possible. |
metadata.dc.relation.ispartof: | MedEspera 2024 |
URI: | https://ibn.idsi.md/collection_view/3104 http://repository.usmf.md/handle/20.500.12710/28686 |
ISBN: | 978-9975-3544-2-4 |
Appears in Collections: | MedEspera 2024
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