Abstract:
Introduction. Seizures are a known complication of stroke. They may increase the cerebral
lesions and induce epileptic status or encephalopathy. The correlation between brain structural
damage, epileptic foci, antiepileptic drugs and clinical outcome is unknown. Late-onset seizures
are thought to be caused by gliosis and the development of a meningocerebral cicatrix. Changes
in membrane properties, deafferentation, selective neuronal loss, and collateral sprouting may
result in hyperexcitability and neuronal synchrony strong enough to cause seizures. Can we
consider as Epilepsy one grand-mal seizure after a massive ischemic stroke?
